Abdominal-Pelvic Imaging
eBook - ePub

Abdominal-Pelvic Imaging

200 Cases (Common Diseases): US, CT and MRI

  1. 218 pages
  2. English
  3. ePUB (mobile friendly)
  4. Available on iOS & Android
eBook - ePub

Abdominal-Pelvic Imaging

200 Cases (Common Diseases): US, CT and MRI

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Table of contents
Citations

About This Book

Maybe we have an obsession for cases, but when we were resident in radiology, we loved to learn especially from cases not only because they are short, exciting and fun, similar to a detective story in which the aim is to get to 'the bottom' of the case, but also because, in the end, that's what radiologists are faced with during their daily work.
The topics covered in the book represent the common and important diseases encountered in abdominal and pelvic imaging. The material presented for each case provides a thorough and comprehensive description of the disease entity, enabling the radiologist or the clinician to develop a clear concept of the entity through the different imaging modalities that are present. What is interesting in this book is one case per page. The book can be used as a means of rapid revision of a large number of cases in a short time or as test of knowledge by masking the radiological description and diagnosis and trying by using the clinical data and radiological images to describe first the pathology then propose a diagnosis.

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Yes, you can access Abdominal-Pelvic Imaging by Ammar Haouimi in PDF and/or ePUB format, as well as other popular books in Medicine & Radiology, Radiotherapy & Nuclear Medicine. We have over one million books available in our catalogue for you to explore.

Information

Case 1

Clinical Presentation
A 36-year-old woman with history of abdominal pain and mild abdominal distension. The liver was enlarged on clinical examination. The abdominal ultrasound revealed an enlarged heterogeneous liver.
Radiological Findings
11
  • Enhanced CT scan, axial (A, B) and MR scan, axial T1 (C), axial T2 fat saturation (D, E) and post-contrast axial T1 (F) images. On CT images, the liver is enlarged with hypertrophied caudate lobe: mottled enhancement with prominent enhancement of the central liver and decreased enhancement of the liver periphery (nutmeg liver). The areas of decreased enhancement are due to decreased portal flow, hepatic congestion and ischemia. Non-visualisation of the hepatic veins. The MR sequences show the same appearances with homogeneous liver on T2 and enhancement of the central liver (the caudate lobe enhances normally as it has a separate draining vein directly into the inferior vena cava) with decreased signal intensity of the atrophied liver periphery. The IVC is patent. Note mild ascites.

Diagnosis: Budd Chiari Syndrome.

Case 2

Clinical Presentation
A 61-year-old man known diabetic type 2 presented with 1-year history of paroxysmal attacks of palpitation, dizziness and headache. The physical examination was normal except the blood pressure which was high during paroxysmal attack (systolic BP varies from 150 to 230 mmHg and diastolic BP varies from 110 to
140 mmHg). The routine laboratory investigations were within normal limits except the glycaemia at 200 mg/Dl. The serum catecholamine level was not done.
Radiological Findings
2
  • Enhanced abdominal CT scan, post-contrast arterial phase axial (A, B) / coronal and sagittal reconstruction (C, D, E) and post-contrast portal phase axial (F) images reveal a well-defined lobulated right suprarenal mass with intense and heterogeneous enhancement and prominent surrounding vessels on arterial phase and rapid washout on portal phase. Note the IVC is displaced anteriorly and the upper pole of the kidney posteriorly. The left adrenal gland appears normal in size and shape (arrow image A).
Diagnosis: Adrenal Pheochromocytoma

Case 3

Clinical Presentation
A 60-year-old man complaining of right upper quadrant pain with abdominal distension after meals. He had a history of surgery for hydatid cysts of the liver 10 years ago. The physical examination was normal except moderate splenomegaly. The routine laboratory investigations were within normal limits.
Radiological Findings
3
  • Pre-(A, B) and post-contrast (C, D, E) abdominal CT scan with coronal reconstruction (F) images showing a dysmorphic liver (past history of surgery for hydatid cysts) with hydatid cysts located in the segments, VIII/VII (CE4 and CE3A, WHO classification) and V (CE3 A, WHO classification). The portal vein and its right branch are dilated, containing germinative membranes and other cyst contents with evidence of fistulous tract between the hydatid cyst located in the segment VIII and dilated right branch of portal vein (arrow in F). Note the presence of tortuous venous structures around the thrombosed portal vein indicating portal cavernoma. The spleen was moderately enlarged with dilated splenic vein (extra-hepatic portal hypertension).
Diagnosis: Hepatic Hydatid Cyst ruptured into the Portal Vein

Case 4

Clinical Presentation
A 40-year-old man presented with acute onset of severe lower abdominal pain, nausea and vomiting with a palpable mid-pelvic mass.
Radiological Findings
4
  • Enhanced Abdominal CT scan, axial (A, B, C) with coronal / sagittal reconstruction (D, E, F) images showing a sausage-shaped mid-pelvic mass with central area containing mesenteric fat and vessels indicating intussusception. Distal within this mass there is a well-defined fatty-density lesion (arrow images, C, E and F) representing a lipoma. Note proximal small bowel dilatation.
Diagnosis: Ileo-ileal Intussusception Secondary to a Lipoma

Case 5

Clinical Presentation
A 35-year-old woman, with history of dyspareunia and severe dysmenorrhoea for 5 years.
Radiological Findings
5
  • MR scan, axial T1 (A), T1 fat saturation (B, C), axial T2 (D) and post-contrast axial / sagittal T1 fat saturation (E, F) images showing multiple and bilateral well-circumscribed contiguous ovarian cystic lesions of high signal intensity on T1 and T1 fat saturation, low signal intensity on T2 with no peripheral enhancement after gadolinium administration (endometriomas). Both ovaries are closed up due to interovarian adhesions (Kissing ovaries). Note that the left ovary shows also another cystic lesion of high signal intensity on T1 and T2 with peripheral regular enhancement after gadolinium administration (haemorrhagic cyst, arrow in E and F). The uterus was normal.
Diagnosis: Bilateral Ovarian Endometriomas with Coexisting Left Haemorrhagic Cyst

Case 6

Clinical Presentation
A 46-year-old woman presented with an abdominal discomfort. An abdominal ultrasound revealed a midline liver with no spleen seen in the left hypochondrium. No significant medical or surgical history.
Radiological Findings
6
  • Enhanced abdominal CT scan, axial (A, B, C, D) with coronal reformatted (E, F) images. The liver is in midline (or bridging liver) with stomach on the right side. Three splenules are seen on the right below the liver (arrows in C and E) with no splenic structure seen in the left hypochondrium. Note an interruption of the inferior vena cava with azygous continuation of the IVC (arrow in A). The head and the corporeo-caudal region of the pancreas are on the right, giving the appearance of a lying V (image C) with Preduodenal position of the portal vein. The duodenojejunal junction does not cross the midline; it should be located on the same side of the body as the stomach and approximately at the level of the duodenal bulb, indicating associated intestinal malrotation.
Diagnosis: Heterotaxy-Polysplenia-Azygous Continuation of IVC

Case 7

Clinical Presentation
A 37-year-old man operated 2 years ago for cerebellar tumour presented with headache, tachycardia, decreased level of consciousness and uncontrolled high blood pressure.
Radiol...

Table of contents

  1. Abdominal-Pelvic Imaging
  2. About the Author
  3. Dedication
  4. Copyright Information ©
  5. Acknowledgements
  6. Foreword
  7. Preface
  8. Case 1
  9. Case 2
  10. Case 3
  11. Case 4
  12. Case 5
  13. Case 6
  14. Case 7
  15. Case 8
  16. Case 9
  17. Case 10
  18. Case 11
  19. Case 12
  20. Case 13
  21. Case 14
  22. Case 15
  23. Case 16
  24. Case 17
  25. Case 18
  26. Case 19
  27. Case 20
  28. Case 21
  29. Case 22
  30. Case 23
  31. Case 24
  32. Case 25
  33. Case 26
  34. Case 27
  35. Case 28
  36. Case 29
  37. Case 30
  38. Case 31
  39. Case 32
  40. Case 33
  41. Case 34
  42. Case 35
  43. Case 36
  44. Case 37
  45. Case 38
  46. Case 39
  47. Case 40
  48. Case 41
  49. Case 42
  50. Case 43
  51. Case 44
  52. Case 45
  53. Case 46
  54. Case 47
  55. Case 48
  56. Case 49
  57. Case 50
  58. Case 51
  59. Case 52
  60. Case 53
  61. Case 54
  62. Case 55
  63. Case 56
  64. Case 57
  65. Case 58
  66. Case 59
  67. Case 60
  68. Case 61
  69. Case 62
  70. Case 63
  71. Case 64
  72. Case 65
  73. Case 66
  74. Case 67
  75. Case 68
  76. Case 69
  77. Case 70
  78. Case 71
  79. Case 72
  80. Case 73
  81. Case 74
  82. Case 75
  83. Case 76
  84. Case 77
  85. Case 78
  86. Case 79
  87. Case 80
  88. Case 81
  89. Case 82
  90. Case 83
  91. Case 84
  92. Case 85
  93. Case 86
  94. Case 87
  95. Case 88
  96. Case 89
  97. Case 90
  98. Case 91
  99. Case 92
  100. Case 93
  101. Case 94
  102. Case 95
  103. Case 96
  104. Case 97
  105. Case 98
  106. Case 99
  107. Case 100
  108. Case 101
  109. Case 102
  110. Case 103
  111. Case 104
  112. Case 105
  113. Case 106
  114. Case 107
  115. Case 108
  116. Case 109
  117. Case 110
  118. Case 111
  119. Case 112
  120. Case 113
  121. Case 114
  122. Case 115
  123. Case 116
  124. Case 117
  125. Case 118
  126. Case 119
  127. Case 120
  128. Case 121
  129. Case 122
  130. Case 123
  131. Case 124
  132. Case 125
  133. Case 126
  134. Case 127
  135. Case 128
  136. Case 129
  137. Case 130
  138. Case 131
  139. Case 132
  140. Case 133
  141. Case 134
  142. Case 135
  143. Case 136
  144. Case 137
  145. Case 138
  146. Case 139
  147. Case 140
  148. Case 141
  149. Case 142
  150. Case 143
  151. Case 144
  152. Case 145
  153. Case 146
  154. Case 147
  155. Case 148
  156. Case 149
  157. Case 150
  158. Case 151
  159. Case 152
  160. Case 153
  161. Case 154
  162. Case 155
  163. Case 156
  164. Case 157
  165. Case 158
  166. Case 159
  167. Case 160
  168. Case 161
  169. Case 162
  170. Case 163
  171. Case 164
  172. Case 165
  173. Case 166
  174. Case 167
  175. Case 168
  176. Case 169
  177. Case 170
  178. Case 171
  179. Case 172
  180. Case 173
  181. Case 174
  182. Case 175
  183. Case 176
  184. Case 177
  185. Case 178
  186. Case 179
  187. Case 180
  188. Case 181
  189. Case 182
  190. Case 183
  191. Case 184
  192. Case 185
  193. Case 186
  194. Case 187
  195. Case 188
  196. Case 189
  197. Case 190
  198. Case 191
  199. Case 192
  200. Case 193
  201. Case 194
  202. Case 195
  203. Case 196
  204. Case 197
  205. Case 198
  206. Case 199
  207. Case 200