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Edhf 2000
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Understanding the nature and role of endothelium-derived hyperpolarizing factor appears to be crucial in the quest for improved treatments for hypertension, diabetes, ischemia-reperfusion and other vascular disorders.
EDHF 2000 comprises the proceedings of the Third International Symposium on endothelium-dependent hyperpolarizations. The first t
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Table of contents
- Cover
- Half Title
- Title Page
- Copyright Page
- Contents
- Preface
- Acknowledgements
- Contributors
- 1 What do gap junctions do anyway?
- 2 Cardiovascular gap junctions : functional diversity, complementation and specialization of connexins
- 3 Endothelium and smooth muscle pathways for conduction along resistance microvessels
- 4 Membrane potential and calcium responses evoked by acetylcholine in submucosal arterioles of the guinea-pig small intestine
- 5 The effects of ouabain, 18α-glycyrrhetinic acid and connexin-mimetic peptides on intercellular communication in cells expressing a Cx43-GFP chimeric protein
- 6 Role of gap junctions in endothelium-dependent hyperpolarizations
- 7 Heterogeneity of EDHF-type relaxations of rabbit and rat arteries analysed with peptides homologous to the extracellular loops of connexins 37, 40 and 43
- 8 Myoendothelial and circumferential spread of endothelium-dependent hyperpolarization in coronary arteries
- 9 Direct myoendothelial contacts in human pulmonary microvessels
- 10 Role of gap junctional communication in EDHF-mediated responses and mechanisms of K+-induced dilatations
- 11 Comparison of α and β isoforms of glycyrrhetinic acid and carbenoxolone as inhibitors of EDHF-type relaxation
- 12 Inhibitory effect of 18-glycyrrhetinic acid on the relaxation induced by acetylcholine in the rat aorta
- 13 A central role for endothelial cell potassium channels in EDHF-mediated responses
- 14 Could the EDHF be K+ in porcine coronary arteries?
- 15 Nitro-L-arginine/indomethacin-resistant relaxations to acetylcholine in small gastric arteries of the rat: Effect of ouabain plus Ba2+ and relation to potassium ions
- 16 Effects of barium, ouabain and K+ on the resting membrane potential and endothelium-dependent responses in rat arteries
- 17 EDHF and potassium : blockade of chloride channels reveals relaxations of rat mesenteric artery to potassium
- 18 Cytochrome P450 2C - a source of EDHF and reactive oxygen species in the porcine coronary artery
- 19 Cortisol increases EDHF-mediated relaxations in porcine coronary arteries and up-regulates the expression of cytochrome P450 2C9
- 20 An arachidonic acid metabolite(s) produced by the endothelial cytochrome P450 isoform, CYP3A4, relaxes the lingual artery of the monkey via K+ channel opening
- 21 EDHF-mediated responses induced by bradykinin in the porcine coronary artery
- 22 Epoxyeicosatrienoic acid release mediates nitric oxide-independent dilatation of rat mesenteric vessels
- 23 Epoxyeicosatrienoic acid and endothelium-dependent hyperpolarization in porcine coronary arteries
- 24 Lipoxygenase-derived metabolites of arachidonic acid are not involved in the endothelium-dependent hyperpolarization to acetylcholine in the carotid artery of the guinea-pig
- 25 Cytochrome P450 isoforms in the brain encode cell specific hy perpolarizing factors with a common mechanism of action
- 26 Expression of recombinant cytochrome P450 epoxygenase in rat brain
- 27 Identification of 11,12,15-trihydroxyeicosatrienoic acid as the mediator of acetylcholine-and arachidonic acid-induced relaxations in the rabbit aorta
- 28 Identification of hydrogen peroxide as an endothelium-derived hyperpolarizing factor in mice
- 29 Components of the potassium currents underlying the actions of endothelium-derived hyperpolarizing factor in arterioles
- 30 Evidence for relaxation to endothelium-derived hyperpolarizing factor (EDHF) in isolated small mesenteric arteries of the mouse
- 31 Pharmacological characterization of potassium channels in intact mesenteric arteries and single smooth muscle cells from eNOS -/- and +/+ mice
- 32 EDHF, which is not NO, is a major endothelium-dependent vasodilator in mice
- 33 Prostacyclin and iloprost in the isolated carotid artery of the guinea-pig
- 34 Role of charybdotoxin/apamin sensitive K+Ca channels in pulsatile perfusion-mediated coronary vasodilatation in vivo
- 35 Essential role of estrogen in the EDHF-mediated responses of mesenteric arteries from middle-aged female rats: possible contribution of gap junctional protein connexin43
- 36 Endothelium-derived hyperpolarizing factor (EDHF) and utero-feto-placental circulation in the rat
- 37 Endothelium-derived hyperpolarizing factor maintains a normal relaxation to bradykinin despite impairment of the nitric oxide pathway in porcine coronary arteries with regenerated endothelium
- 38 Mechanisms underlying the vasodilatation caused by bradykinin in essential hypertensive patients
- 39 Influence of diabetes on endothelium-dependent responses in mesenteric and femoral arteries of rats
- 40 Folate restores the NO synthase-and cyclooxygenase-resistant renal vasodilator response to acetylcholine in diabetes
- 41 Resistance of EDHF-mediated relaxations to oxidative stress in human radial arteries
- 42 Critical limb ischemia results in different types of endothelial dysfunction depending on the vascular bed studied
- 43 Potentiated EDHF-mediated dilatations in the rat middle cerebral artery following ischemia/reperfusion
- 44 The EDHF-dependent but not the NO-dependent component of the acetylcholine-induced relaxation of the rabbit aorta is resistant to ionized radiation
- 45 Inhibition of converting enzyme prevents the age-related decline in endothelium-dependent hyperpolarization
- 46 Effects of a converting enzyme inhibitor, an AT1-receptor antagonist and their combination on endothelial dysfunction in hypertension
- 47 EDHF: gap junction or chemical? and many other questions
- References
- Index